This is a discussion on Ask here for fulltext research papers (Warning: WOT) within the Research Articles/User Guides forums, part of the Prohormone Forum category; Relative Potency of Various Steroids in an Anabolic-Androgenic Assay Using the Castrated Rat...
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I start to remember. Ormonoterapia Richter (Milano) invented Roxilon (dimethazine)Originally Posted by BrYmAsTeR17
Un nuovo steroide avente azione proteanabolica: La Dimetazina
I have that paper somewhere but I must find it.
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bmsimon do you still have this one? I seem to have lost the copy you gave me.
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Sync 4shared.
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In 4shared.
I don't have access to this one.
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cool, thanks.
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A tad unrelated but really need this:
I am looking for chapter 3 by O’Donovan. Genetic toxicity testing to qualify alerting impurities.
In “Genotoxic Impurities: Strategies for Identification and Control”. Edited by A. Teasdale, John Wiley & Sons, Inc, Hoboken NJ, pp 67-96 (2010).
I usually don't have too much trouble, even with obscure titles, but this one has eluded me. Hopefully someone can help, google scholar has a 'preview' however the pages it leaves out are the ones I need most. Thanks in advance if anyone can find it.
Cynical realism is the intelligent man's best excuse for doing nothing in an intolerable situation. -Aldous Huxley
Anyone get this for me?
Curr Vasc Pharmacol. 2011 Jul 1;9(4):385-400.
Pleiotropic effects of nicotinic acid: beyond high density lipoprotein cholesterol elevation.
Florentin M, Liberopoulos EN, Kei A, Mikhailidis DP, Elisaf MS.
Source
Department of Clinical Biochemistry (Vascular Disease Prevention Clinics), Royal Free Campus, University College London Medical School, University College London (UCL), UK.
Abstract
AIMS:
Treatment with statins has significantly reduced cardiovascular morbidity and mortality, an effect attributed to both the low-density lipoprotein cholesterol (LDL-C) lowering capacity and the pleiotropic actions of these drugs. However, residual risk remains even after intense LDL-C lowering. Therefore, additional treatment with lipid-lowering drugs which improve other lipid parameters and have favourable non-lipid effects may be of clinical value. The aim of the present article is to review the actions of nicotinic acid and comment on the limitations and possible benefits of this drug in clinical practice.
METHODS:
Relevant articles were identified through a Pubmed search up to July 2010.
RESULTS:
Nicotinic acid (niacin) improves the lipid profile and has been associated with reduction in morbidity and mortality from cardiovascular disease. This favourable outcome may be due to several beneficial actions of this drug, such as antithrombotic, anti-inflammatory and antioxidant. However, its use has been limited due to side effects, especially flushing. A novel formulation with a prostaglandin D2 receptor antagonist (laropiprant) appears to substantially decrease the frequency and intensity of flushing, without affecting the other properties of niacin. Some concerns regarding treatment with nicotinic acid include impaired glucose metabolism and elevations in uric acid and homocysteine levels.
CONCLUSION:
Nicotinic acid is a safe supplementary (to statins) lipid lowering agent which may also improve cardiovascular outcomes. Whether its combination with laropiprant will be proved equally effective and more favourable in terms of adverse effects remains to be established by large clinical trials.
PMID: 21314635 [PubMed - indexed for MEDLINE]
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Sorry.
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Thank you.
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Repped for trying
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Thank you all.
Guys you are doing a wonderful job...
You using these texts for something?
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While less than enthusiastic about nootropics, this one's pronounced CNS stimulating properties I find interesting.
http://www.totalflexblog.com/articles/phenylpiracetam/
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Bmison... you are a god, reps for life, thanks a lot man.
Cynical realism is the intelligent man's best excuse for doing nothing in an intolerable situation. -Aldous Huxley
You using these texts for something?
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Purely for personal education. Maybe I am an old, rusty guru that needs a kind of cognitive refreshment, occasionally.
Beware! I will be asking for some research papers soon.
I apologize if that sounded rude, didn't mean for it to come off that way.
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no problem bro. i see
A few interesting stuff on designer pharmacology:
Detection and pharmacokinetics of tetrahydrogestrinone in horses
Tetrahydrogestrinone analysis and designer steroids revisited.
The use of in vitro technologies and high-resolution/accurate-mass LC-MS to screen for metabolites of 'designer' steroids in the equine
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Thanks Henry for those 2 artikels.
The stubborn metabolic behaviour of the THG molecule is proved once again.
My theory was right.
Such an unfriendly xenobiotic!
Many 4,9,11-triene steroids are similar in this regard, at least in vivo.
The fact that they detected some reduced metabolites though is cool(although low abundance), just wish we could find out which double-bond(s) were reduced.
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^referring to 2011 study
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